Spinal Decompression Research Information

Spinal Decompression is a drug-free and non-surgical answer for disc related syndromes of the lumbar or cervical spine. Many people across the country have found relief from the pain associated with herniated discs, bulging discs, facet syndrome, degenerative joint disease, pinched nerves, and other spinal afflictions.Spinal Disc Decompression uses computer-aided technology to apply gentle, non-surgical decompression to your spine, increasing circulation into the spinal discs and joints, thus helping to relieve the symptoms that cause pain and dysfunction.We use the exclusive DRX 9000 machine in our office. The best Non Surgical Spinal Decompression table on the Market.

Why Spinal Disc Decompression Works:

This FDA cleared technology relieves pain by enlarging the space between the discs. The negative pressure of decompression releases pressure that has built on the disc and nerves, allowing the herniated and bulging disc to eventually return to a normal position. Decompression is truly the most effective for severe cases of herniation, degeneration, arthritis, stenosis and pressure on the nerve root. In 2004, Orthopedic Technological Review performed a study finding 86% of all cases experience spinal pain relief with disc decompression.

What is a Disc? What does a Disc do?

The disc consisting of a soft cartilage material that separates the spinal vertebrae. We have Discs starting at the the third cervical all the way down to the L 5, which is the lowest Lumbar vertebrae . The middle of the Disc is called the nucleus and the outer layer is called the annulus.These discs act as shock absorbers and after we are done growing usually around 18 - 19 years old, become avascualr (absence of blood vessels). One of the reasons that our discs give us so many problems is that they do not heal well without a good blood supply. When we break a bone it heals quickly and is actually stronger than before! Our discs are avascular so if a disc is injured it continues to degenerate and does not heal properly. One of the amazing benefits of decompression is that it heightens the disc space, and returns it to their normal size and function.

The Decompression therapy also causes the disc to heal because the treatment stimulates the cartildge to regenerate, an act the disc cannot do on its own without the decompression treatment.Spinal Decompression also causes the nutrient rich blood to fill the disc space which promotes the healing response.

Bulging Disc

The term bulging disc (Disc Herniation) implies that a rupture or a tear of the ring shaped fibers occurs, allowing the spread of nucleur material beyond the verterbral margin. The nuclear material may protrude out and cause a swelling of the outer annulus fibrosis or rupture through the annulus and push behind the back ligament.

Four classifications that describe the different disc lesions.

  1. Annular bulge
  2. Protrusion (Herniation)
  3. Extrusion
  4. Free Disc Fragment (Sequestration)

The annular bulge or Disc bulge is a small disc herniation that does not directly contact the nerve root and left untreated can easily progress to a larger degree of nuclear protrusion because of the loss of the annular fibers to contain the nucleus. Degeneration will predispose annular fibers to failure following trauma.

Disc Protrusion (Herniation) A Disc herniation represents a rupture of nuclear material through a defect in annulus, producing a focal extension of the disc or a broad based extension of the disc margin.Intervertebral disc herniation result in some degree from central canal or foraminal occlusion.

Disk Extrusion applies when a portion of the nucleus pulposus fibrocartilage and end plate cartilage have migrated through comprised outer annular fibers. Disc extrusions may compress the root, cord or both and can cause myeloradiculopathy (shooting pain).

Free disc Fragment (Sequestered Disc) refers to the extra-annular separation and migration of a piece of nuclear material. Cauda Equina syndrome are common with intradural migration. This is when you lose control of bowel and bladder function. This is a emergency situation and usually requires surgery to correct the problem.

The most common area is the Lower Lumbar vertebra L5. The pressure causes the jelly material inside the disc to "bulge" or "slip" out of place .The bulge itself puts pressure on the nerves( usually the spinal nerve).The Spinal nerve is very sensitive even a small amount of pressure causes the nerve to dysfunction. The most common cervical disc to herniate is also the fifth and can cause cervicobrachial syndrome or symptoms like carpal tunnel syndrome or weakness in the arms or a burning sensation in the arms.

Disc Dessication is when the annular fibers degenerate due to dehydration, One preventative step is to drink as much water as can if you feel that you have or are developing a Disc Herniation.

Leg Pain or Sciatica

Pain in legs like Sciatica

The Sciatic nerve is the largest nerve in the body and it is a common problem with people and when a disc herniates it can causes Sciatica which can feel like a sharp shooting pain down the look into the buttocks or even into the toes. In extreme examples it can cause permanent numbness and loss of muscle strength.

Decompression Technology

NASA does not endorse any equipment or table nor do they claim that this was their discovery.

Please be careful when finding a doctor that claims that the DRX 9000 is the only table that provides true spinal decompression, this is only a marketing gimmick and completely false. The FDA cleared several tables to be called Spinal Disc decompression and they all do basically the same thing some have fancier options. Some of these tables have names like the spine med, the lordex machine, ABS, Extentrac, 3D active trac. In my practice I use the DRX 9000 unit because I believe it utilizes the latest technological advances and is the most comfortable.

Spinal Decompression

By Thomas A. Gionis, MD, JD, MBA, MHA, FICS, FRCS, and Eric Groteke, DC, CCIC

The outcome of a clinical study evaluating the effect of nonsurgical intervention on symptoms of spine patients with herniated and degenerative disc disease is presented.

This clinical outcomes study was performed to evaluate the effect of spinal decompression on symptoms and physical findings of patients with herniated and degenerative disc disease. Results showed that 86% of the 219 patients who completed the therapy reported immediate resolution of symptoms, while 84% remained pain-free 90 days post-treatment. Physical examination findings showed improvement in 92% of the 219 patients, and remained intact in 89% of these patients 90 days after treatment. This study shows that disc disease, the most common cause of back pain, which costs the American health care system more than $50 billion annually can be cost-effectively treated using spinal decompression. The cost for successful non-surgical therapy is less than a tenth of that for surgery. These results show that biotechnological advances of spinal decompression reveal promising results for the future of effective management of patients with disc herniation and degenerative disc diseases. Long-term outcome studies are needed to determine if non-surgical treatment prevents later surgery, or merely delays it.


With the recent advances in biotechnology, spinal decompression has evolved into a cost-effective nonsurgical treatment for herniated and degenerative spinal disc disease, one of the major causes of back pain. This nonsurgical treatment for herniated and degenerative spinal disc disease works on the affected spinal segment by significantly reducing intradiscal pressures.1 Chronic low back pain disability is the most expensive benign condition that is medically treated in industrial countries. It is also the number one cause of disability in persons under age 45. After 45, it is the third leading cause of disability.2 Disc disease costs the health care system more than $50 billion a year.

The intervertebral disc is made up of sheets of fibers that form a fibrocartilaginous structure, which encapsulates the inner mucopolysaccharide gel nucleus. The outer wall and gel act hydrodynamically. The intrinsic pressure of the fluid within the semirigid enclosed outer wall allows hydrodynamic activity, making the intervertebral disc a mechanical structure.3 As a person utilizes various normal ranges of motion, spinal discs deform as a result of pressure changes within the disc.4 The disc deforms, causing nuclear migration and elongation of annular fibers. Osteophytes develop along the junction of vertebral bodies and discs, causing a disease known as spondylosis. This disc narrows from the alteration of the nucleus pulposus, which changes from a gelatinous consistency to a more fibrous nature as the aging process continues. The disc space thins with sclerosis of the cartilaginous end plates and new bone formation around the periphery of the contiguous vertebral surfaces. The altered mechanics place stress on the posterior diarthrodial joints, causing them to lose their normal nuclear fulcrum for movement. With the loss of disc space, the plane of articulation of the facet surface is no longer congruous. This stress results in degenerative arthritis of the articular surfaces.5

This is especially important in occupational repetitive injuries, which make up a majority of work-related injuries. When disc degeneration occurs, the layers of the annulus can separate in places and form circumferential tears. Several of these circumferential tears may unite and result in a radial tear where the material may herniate to produce disc herniation or prolapse. Even though a disc herniation may not occur, the annulus produces weakening, circumferential bulging, and loss of intervertebral disc height. As a result, discograms at this stage usually reveal reduced interdiscal pressure.

The early changes that have been identified in the nucleus pulposus and annulus fibrosis are probably biomechanical and relate to aging. Any additional trauma on these changes can speed up the process of degeneration. When there is a discogenic injury, physical displacement occurs, as well as tissue edema and muscle spasm, which increase the intradiscal pressures and restrict fluid migration.6 Additionally, compression injuries causing an endplate fracture can predispose the disc to degeneration in the future.

The alteration of normal kinetics is the most prevalent cause of lower back pain and disc disruption and thus it is vital to maintain homeostasis in and around the spinal disc; Yong-Hing and Kirkaldy-Willis7 have correlated this degeneration to clinical symptoms. The three clinical stages of spinal degeneration include:

  1. Stage of Dysfunction. There is little pathology and symptoms are subtle or absent. The diagnosis of Lumbalgia and rotatory strain are commonly used.
  2. Stage of Instability. Abnormal movement of the motion segment of instability exists and the patient complains of moderate symptoms with objective findings. Conservative care is used and sometimes surgery is indicated.
  3. Stage of Stabilization. The third phase where there are severe degenerative changes of the disc and facets reduce motion with likely stenosis.

Spinal decompression has been shown to decompress the disc space, and in the clinical picture of low back pain is distinguishable from conventional spinal traction.8,9 According to the literature, traditional traction has proven to be less effective and biomechanically inadequate to produce optimal therapeutic results.8-11 In fact, one study by Mangion et al concluded that any benefit derived from continuous traction devices was due to enforced immobilization rather than actual traction.10 In another study, Weber compared patients treated with traction to a control group that had simulated traction and demonstrated no significant differences.11 Research confirms that traditional traction does not produce spinal decompression. Instead, decompression, that is, unloading due to distraction and positioning of the intervertebral discs and facet joints of the lumbar spine, has been proven an effective treatment for herniated and degenerative disc disease, by producing and sustaining negative intradiscal pressure in the disc space. In agreement with Nachemon´s findings and Yong-Hing and Kirkaldy-Willis,1 spinal decompression treatment for low back pain intervenes in the natural history of spinal degeneration.7,12 Matthews13 used epidurography to study patients thought to have lumbar disc protrusion. With applied forces of 120 pounds x 20 minutes, he was able to demonstrate that the contrast material was drawn into the disc spaces by osmotic changes. Goldfish14 speculates that the degenerated disc may benefit by lowering intradiscal pressure, affecting the nutritional state of the nucleus pulposus. Ramos and Martin8 showed by precisely directed distraction forces, intradiscal pressure could dramatically drop into a negative range. A study by Onel et al15 reported the positive effects of distraction on the disc with contour changes by computed tomography imaging. High intradiscal pressures associated with both herniated and degenerated discs interfere with the restoration of homeostasis and repair of injured tissue.

Biotechnological advances have fostered the design of Food and Drug Administration-approved ergonomic devices that decompress the intervertebral discs. The biomechanics of these decompression/reduction machines work by decompression at the specific disc level that is diagnosed from finding on a comprehensive physical examination and the appropriate diagnostic imaging studies. The angle of decompression to the affected level causes a negative pressure intradiscally that creates an osmotic pressure gradient for nutrients, water, and blood to flow into the degenerated and/or herniated disc thereby allowing the phases of healing to take place.

This clinical outcomes study, which was performed to evaluate the effect of spinal decompression on symptoms of patients with herniated and degenerative disc disease, showed that 86% of the 219 patients who completed therapy reported immediate resolution of symptoms, and 84% of those remained pain-free 90 days post-treatment. Physical examination findings revealed improvement in 92% of the 219 patients who completed the therapy.


The study group included 229 people, randomly chosen from 500 patients who had symptoms associated with herniated and degenerative disc disease that had been ongoing for at least 4 weeks. Inclusion criteria included pain due to herniated and bulging lumbar discs that is more than 4 weeks old, or persistent pain from degenerated discs not responding to 4 weeks of conservative therapy. All patients had to be available for 4 weeks of treatment protocol, be at least 18 years of age, and have an MRI within 6 months. Those patients who had previous back surgery were excluded. Of note, 73 of the patients had experienced one to three epidural injections prior to this episode of back pain and 22 of those patients had epidurals for their current condition. Measurements were taken before the treatments began and again at week two, four, six, and 90 days post treatment. At each testing point a questionnaire and physical examination were performed without prior documentation present in order to avoid bias. Testing included the Oswetry questionnaire, which was utilized to quantify information related to measurement of symptoms and functional status. Ten categories of questions about everyday activities were asked prior to the first session and again after treatment and 30 days following the last treatment.

Testing also consisted of a modified physical examination, including evaluation of reflexes (normal, sluggish, or absent), gait evaluation, the presence of kyphosis, and a straight leg raising test (radiating pain into the lower back and leg was categorized when raising the leg over 30 degrees or less is considered positive, but if pain remained isolated in the lower back, it was considered negative). Lumbar range of motion was measured with an ergonometer. Limitations ranging from normal to over 15 degrees in flexion and over 10 degrees in rotation and extension were positive findings. The investigator used pinprick and soft touch to determine the presence of gross sensory deficit in the lower extremities.

Of the 229 patients selected, only 10 patients did not complete the treatment protocol. Reasons for noncompletion included transportation issues, family emergencies, scheduling conflicts, lack of motivation, and transient discomfort. The patient protocol provided for 20 treatments of spinal decompression over a 6-week course of therapy. Each session consisted of a 45-minute treatment on the equipment followed by 15 minutes of ice and interferential frequency therapy to consolidate the lumbar paravertebral muscles. The patient regimen included 2 weeks of daily spinal decompression treatment (5 days per week), followed by three sessions per week for 2 weeks, concluding with two sessions per week for the remaining 2 weeks of therapy.

On the first day of treatment, the applied pressure was measured as one half of the person´s body weight minus 10 pounds, followed on the second day with one half of the persons body weight. The pressure placed for the remainder of the 18 sessions was equivalent to one half of the patient´s body weight plus an additional 10 pounds. The angle of treatment was set according to manufacturer´s protocol after identifying a specific lumbar disc correlated with MRI findings. A session would begin with the patient being fitted with a customized lower and upper harness to fit their specific body frame. The patient would step onto a platform located at the base of the equipment, which simultaneously calculated body weight and determined proper treatment pressure. The patient was then lowered into the supine position, where the investigator would align the split of table with the top of the patient´s iliac crest. A pneumatic air pump was used to automatically increase lordosis of the lumbar spine for patient comfort. The patient´s chest harness was attached and tightened to the table. An automatic shoulder support system tightened and affixed the patient´s upper body. A knee pillow was placed to maintain slight flexion of the knees. With use of the previously calculated treatment pressures, spinal decompression was then applied. After treatment, the patient received 15 minutes of interferential frequency (80 to 120 Hz) therapy and cold packs to consolidate paravertebral muscles.

During the initial 2 weeks of treatment, the patients were instructed to wear lumbar support belts and limit activities, and were placed on light duty at work. In addition, they were prescribed a nonsteroidal, to be taken 1 hour before therapy and at bedtime during the first 2 weeks of treatment. After the second week of treatment, medication was decreased and moderate activity was permitted.

Data was collected from 219 patients treated during this clinical study. Study demographics consisted of 79 female and 140 male patients. The patients treated ranged from 24 to 74 years of age (see Table 1). The average weight of the females was 146 pounds and the average weight of the men was 195 pounds. According to the Oswestry Pain Scale, patients reported their symptoms ranging from no pain (0) to severe pain (5).


The patients were further subdivided into six groups:

  1. single lateral herniation 67 cases
  2. single central herniation 22 cases
  3. single lateral herniation with disc degeneration 32
  4. cases single central herniation with disc degeneration. 24 cases
  5. more than 1 herniation with disc degeneration 17
  6. cases more than 1 herniation without disc degeneration 57 cases


According to the self-rated Oswestry Pain Scale, treatments were successful in 86% of the 219 patients included in this study (Table 2, page 39). Treatment success was defined by a reduction in pain to 0 or 1 on the pain scale. In 188 patients, the perception of pain was 0 to occasional 1 without further need for medication or treatment . These patients reported complete resolution of pain, lumbar range of motion was normalized, and there was recovery of sensory and motor loss. The remaining 31 patients reported significant pain and disability, despite some improvement in their overall pain and disability score.

Diagnosis MRI

No. of CaseS

Female Patients

Male Patients

Positive Result

No Result

% of Success

Single Herniation Lateral







Single Herniation Central







Single Herniation w/ Degeneration







Single Herniation Lateral w/ Degeneration







Multiple Herniations w/o Degeneration







Multiple Herniations w/ Degeneration














Table 2. Results on self-rated Oswestry Pain Scale after treatment.

In this study, only patients diagnosed with herniated and degenerative discs with at least a 4-week onset were eligible. Each patient´s diagnosis was confirmed by MRI findings. All selected patients reported 3 to 5 on the pain scale with radiating neuritis into the lower extremities. By the second week of treatment, 77% of patients had a greater than 50% resolution of low back pain. Subsequent orthopedic examinations demonstrated that an increase in spinal range of motion directly correlated with an improvement in straight leg raises and reflex response. Table 2 shows a summary of the subjective findings obtained during this study by category and total results post treatment. After 90 days, only five patients (2%) were found to have relapsed from the initial treatment program.

Diagnosis MRI Findings

Improved Gait

Sluggish toNormalReflexes

Improved Sensory Reception

Improved Motor Limitation

Abnormal toNormal Straight Leg Raise Test

ImprovedSpinal Rangeof Motion

Single Herniation Lateral







Single Herniation Central







Single Herniation w/ Degeneration







Single Herniation Lateral w/ Degeneration







Multiple Herniations w/o Degeneration







Multiple Herniations w/ Degeneration














Table 3. Percentage of patients that had improved physical exam findings post treatment.

Ninety-two percent of patients with abnormal physical findings improved post-treatment. Ninety days later only 3% of these patients had abnormal findings. Table 3 summarizes the percentage of patients that showed improvement in physician examination findings testing both motor and sensory system function after treatment. Gait improved in 96% of the individuals who started with an abnormal gait, while 96% of those with sluggish reflexes normalized. Sensory perception improved in 93% of the patients, motor limitation diminished in 86%, 89% had a normal straight leg raise test who initially tested abnormal, and 90% showed improvement in their spinal range of motion.


What is the difference between traction and decompression?

Many health Professionals specializing in lumbar spine pathology have criticized traditional traction. Traction fails in many cases because it can cause muscular stretch receptors to fire, which then cause spinal muscles to contract. This muscular responses actually causes an increase in intradiscal pressure. On the other hand, true decompression is achieved by gradual and calculated increases of distraction forces to spinal structures, utilizing various degrees of distraction forces.

A highly specialized computer must modulate the application of distraction forces in order to achieve the ideal effect. The system uses applies a gentle, curved angle pull which yields far greater treatment results that a less comfortable, sharp angle pull. Distraction must be offset by cycles of partial relaxation.

The system continuously monitors spinal resistance and adjusts the forces accordingly. A specific lumbar segment can be targeted for treatment by changing the angle of distraction. This patented technique of decompression may prevent muscle spasm. Constant activity monitoring takes place at a rate of 10,000 times per second, making adjustments not perceived by the eye as many as 20 times per second via its fractional metering and monitoring system.

True decompression also involves the use of a special pelvic harness that supports the lumbar spine during therapy. Negative pressure within the disc is maintained throughout the treatment session. Using true decompression, the pressure within the disc space can actually be lowered to about -150 mmHg. As a result, the damaged disc may begin to rehydrate with nutrients and oxygen.

Isn´t decompression just another name for a traction machine?

No. There is a big different between distraction, traction, and decompression. Traction has been around for hundreds of years. The problem with traction, as it is known today, is that it is not always beneficial. In 1998, the Scientific American rated traction to be of little or no value in the examination of effective therapies for lower back pain. This finding is consistent with many studies that report traction can often times signal a painful splinting response and may put a patient's back muscles into spasms, resisting any attempts to effect a change on the disc.

Distraction, a term used to describe a bending of the joint distraction technique, attempts to reposition the spine from the offending lesion. This technique has been shown to be very effective, even though potentially damaging to the person performing the technique and largely dependent on the skill of the technician. Like traction, distraction procedures are limited in the ability to reduce the intradiscal pressure, or produce a negative pressure within the disc encouraging the disc to consume fluid, nutrients and creating an environment for repair.

What Kind of Time Commitments Would I Need To Commit To?

Each treatment session averages 25 to 30 minutes in duration (optimum results are accompished with sessions that incorporate 10 to 15 decompression/relaxation cycles). On average, a daily 20-30 minutes sessionis necessary for patient self-healing to occur.

Herniated discs will generally respond within 20 sessions, while patients with degenerated discs may need ongoing therapy at regulated intervals to continue pain free. Still other patients, due to lifestyle or occupation, may also require maintenance therapy.

How long before I begin to experience change?

Many times a patient experiences some relief within the first few (3-7) treatments. Usually by the 12th to 15th treatment all patients have reported some decrease in symptoms. Patients not showing significant improvement by the 15th to 18th session may be need further diagnostic evaluation.

What are signs and symptoms disc herniation.

If you sneeze, Cough or bear down with pain, this is usually an indication that the disc is involved. Patients can have back pain with or without leg pain and leg pain and weakness without having back pain.

Can I still go to work?

Yes! Many people do it before work or on their lunch hour. You should feel better after each and every session.

Will my insurance cover this treatment?

This is on an individual basis. We will gladly call for authorization to get it covered, if at all possible, while you see the doctor. And, if there is a percentage that will not be covered, we will work with you. We want you to have relief from the chronic pain you have been suffering from.


In conclusion, nonsurgical spinal decompression provides a method for physicians to properly apply and direct the decompressive force necessary to effectively treat discogenic disease. With the biotechnological advances of spinal decompression, symptoms were restored by subjective report in 86% of patients previously thought to be surgical candidates and mechanical function was restored in 92% using objective data. Ninety days after treatment only 2% reported pain and 3% relapsed, by physical examination exhibiting motor limitations and decreased spinal range of motion. Our results indicate that in treating 219 patients with MRI-documented disc herniation and degenerative disc diseases, treatment was successful as defined by: pain reduction; reduction in use of pain medications; normalization of range of motion, reflex, and gait; and recovery of sensory or motor loss. Biotechnological advances of spinal decompression indeed reveal promising results for the future of effective management of patients with disc herniation and degenerative disc diseases. The cost for successful nonsurgical therapy is less than a tenth of that for surgery. Long-term outcome studies are needed to determine if nonsurgical treatment prevents later surgery or merely delays it.

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